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Introduction
Journal of Gannan Medical University is a comprehensive academic journal in the field of medicine and health, sponsored by Gannan Medical University and publicly distributed both domestically and internationally. It is published monthly.
The journal aims to report new achievements, new technologies, and new experiences in scientific research, medical practice, and medical education at our university and beyond, promote academic exchange, and raise the level of scholarship. It primarily publishes original research articles in medicine and health, featuring the following columns: Monographic Studies, Basic Research, Pharmaceutical Sciences, Clinical Research, Medical Imaging and Nuclear Medicine, Public Health and Management, Nursing Research, and Reviews.
To facilitate the exchange of medical information and contribute to the development of China's medical and health services, we welcome submissions from researchers at medical colleges and universities, as well as scientific research, teaching, and medical institutions within the health system across the country.
Sections
Monographic Studies，Basic Research，Pharmaceutical Sciences，Clinical Research，Medical Imaging and Nuclear Medicine，Public Health and Management，Nursing Research，Reviews
Study on the mechanism of metformin regulating osteogenic differentiation of bone marrow mesenchymal stem cells in diabetic rats by up-regulating BMP-2/IGF-1 expression
WANG Yang;GUO Li;KE Ruiqiong;LIU Dengke;WU Dan;Objective : To investigate the effects of different concentrations of metformin on osteogenic differentiation of bone marrow mesenchymal stem cells(BMSCs) in diabetic rats in vitro and its molecular mechanism. Methods : Diabetic male Wistar rat models were established by intraperitoneal injection of streptozotocin(STZ, 40 mg/kg). BMSCs were isolated, cultured, and the third-generation cells were used for the experiments. The cells were randomly divided into three groups: control group(without metformin), low-dose group(10 μmol/L metformin), and high-dose group(100 μmol/L metformin). All groups were cultured in osteogenic induction medium. The alkaline phosphatase(ALP) activity of the cells was detected on the 7 th and 14 th days of induction differentiation, and alizarin red staining was used to observe the formation of calcified nodules. The expression levels of osteogenic-related proteins bone morphogenetic protein-2(BMP-2) and insulin-like growth factor-1(IGF-1) were also detected by protein immunoblotting. Results : On the 7 th and 14 th days of induction differentiation, compared with the control group, the ALP activity and the formation of calcified nodules increased in the low-dose and high-dose metformin groups, and the high-dose group was superior to the low-dose group. Compared with the control group, both low-dose and high-dose groups showed increased protein expression levels of BMP-2 and IGF-1(P<0. 05), with the high-dose group exhibiting higher protein expression levels of BMP-2 and IGF-1 than the low-dose group(P<0. 05). Conclusion : Metformin can dose-dependably enhance the osteogenic differentiation ability of BMSCs in diabetic rats, and the mechanism may be related to the upregulation of BMP-2 and IGF-1 protein expressions.
Predictive value of serum parameters, fetal umbilical artery blood flow parameters, and growth indicators for pregnancy outcomes in pregnant women with gestational diabetes mellitus
CHEN Limin;CHEN Chu;YE Mei;YE Shengfu;Objective : To analyze the predictive value of serum indicators, fetal umbilical artery blood flow parameters and fetal growth indicators for pregnancy outcomes in pregnant women with gestational diabetes mellitus(GDM). Methods : A total of 300 pregnant women with GDM admitted to Guangdong Clifford Hospital from June 2022 to June 2025 were enrolled in this retrospective study. According to pregnancy outcomes, the subjects were divided into adverse outcome group and favorable outcome group. General data, serum indicators, fetal umbilical artery blood flow parameters and fetal growth indicators were collected and compared between the two groups. Logistic regression analysis was performed on the statistically different indicators to identify the independent risk factors for adverse pregnancy outcomes in GDM women. A nomogram and receiver operating characteristic(ROC) curve were drawn to evaluate the predictive value of these factors for adverse pregnancy outcomes in pregnant women with GDM. Results : Among the 300 GDM pregnant women, 46 cases had adverse pregnancy outcomes(adverse outcome group), and the remaining 254 cases had favorable pregnancy outcomes(favorable outcome group). The levels of fasting plasma glucose(FPG), fetal umbilical artery pulsatility index(PI), systolic/diastolic ratio(S/D), abdominal circumference(AC) and estimated fetal weight(EFW) in the adverse outcome group were significantly higher than those in the favorable outcome group(P<0. 05), and these indicators were independent risk factors for adverse pregnancy outcomes in GDM women. The nomogram combined with calibration curve showed that the predictive model established on the above factors had higher application value. The area under the curve(AUC) of the ROC curve of the combined prediction for adverse pregnancy outcomes was 0. 875(95% CI: 0. 821-0. 929). Conclusion : Fasting plasma glucose, PI, S/D, AC and EFW are independent risk factors for adverse pregnancy outcomes in pregnant women with GDM. The nomogram prediction model constructed based on the above indicators has higher predictive value, and combined detection of the five indicators can improve the predictive accuracy of adverse pregnancy outcomes in GDM pregnant women.
The role of polysaccharide from the seed cakes of Camellia oleifera Abel in inhibiting the development of murine colitis
XI Yuling;HE Liangmei;WANG Xiaoyin;LIU Zhiping;Objective : To investigate the preventive and therapeutic effects of polysaccharides from the seed cakes of Camellia oleifera Abel(COCP) on colitis in mice. Methods : C57 BL/6 mice were randomly divided into five groups: a negative control group, a colitis model group, and COCP intervention groups receiving low(25 mg/kg), medium(50 mg/kg) and high(100 mg/kg) doses. An experimental colitis model was induced using 3% sodium dextran sulphate(DSS); COCP was administered prophylactically for 14 consecutive days via gastric lavage, starting from 7 days prior to model induction and continuing until 7 days after induction; the disease activity index(DAI) scores were calculated, and changes in inflammatory cytokines, tight junction molecules(both gene expression and protein levels) in colonic tissue, and faecal microbiota were assessed; bone marrow-derived macrophages(BMDMs) were isolated from C57 BL/6 mice and cultured in vitro. The cells were divided into five groups: a negative control group, an inflammatory model group, and COCP intervention groups at low(3. 9 μg/mL), medium(7. 8 μg/mL) and high(15. 6 μg/mL) concentrations. An in vitro inflammatory model was established by stimulating BMDMs with lipopolysaccharide(LPS); the COCP intervention groups at each concentration were treated with the corresponding concentration of COCP, and the effects of COCP on the BMDM inflammatory model were observed; transcriptomic sequencing was used to analyse the molecular mechanisms of COCP. Results : Compared with the colitis model group, following COCP intervention, the rate of weight loss in mice was markedly slowed, the DAI was significantly reduced, colon length increased, and histopathological damage to the colon was alleviated. Furthermore, COCP downregulated the mRNA expression levels of inflammation-related cytokines(Il6, Tnf and Ccl2) in colonic tissue and inhibited the phosphorylation of NF-κB p65; In an LPS-induced BMDM inflammation model, compared with the inflammation control group, COCP suppressed the gene expression levels of Tnf and Ccl2. Mechanistically, COCP increased the number of goblet cells in colonic tissue and upregulated the gene and protein expression levels of tight junction molecules such as E-cadherin and Occludin; it also modulated the gut microbiota composition, increasing the abundance of beneficial bacteria such as Tannerellaceae and Akkermansiaceae. Transcriptomic sequencing revealed that COCP significantly upregulates the expression of genes such as Gm42031, Rps3a2 and Grm4, whilst downregulating the expression of genes such as Saa1, Gm21887 and Cyp2d26. Conclusion : COCP inhibits colitis in mice, probably exerting its inhibitory effect by suppressing the inflammation level, enhancing the barrier function, regulating the ecology of the flora, and improving the DNA repair ability.
Investigations on the role and mechanisms of Telaglenastat in pathological cardiac hypertrophy
WANG Kun;LIU Ruobing;LI Hongliang;HU Yufeng;Objective : To investigate the therapeutic effect of the small molecule compound Telaglenastat on pathological myocardial hypertrophy, aiming to provide a potential lead compound and research direction for the clinical development of related therapeutic agents. Methods : H9C2 and AC16 cells were treated with different concentrations of Telaglenastat, and the CCK-8 assay was used to evaluate the cytotoxic effects of telaglenastat on normal cardiomyocytes. A phenylephrine(PE)-induced neonatal rat cardiomyocyte hypertrophy model was established, and the cells were divided into four groups(DMSO+PBS, DMSO+PE, 2. 5 μmol/L Telaglenastat+PE, and 5 μmol/L Telaglenastat+PE) for efficacy evaluation. The mRNA and protein expression levels of the cardiac hypertrophy markers atrial natriuretic peptide(Anp) and B-type natriuretic peptide(Bnp) were detected by RT-qPCR and western blot, respectively. Transcriptomic analysis was performed to investigate the effects of telaglenastat on signaling pathways related to cardiac hypertrophy and its underlying mechanisms. A mouse model of transverse aortic constriction(TAC)-induced pathological cardiac hypertrophy was established, and the mice were divided into four groups: sham surgery + vehicle(Sham+Vehicle), sham surgery + Telaglenastat(Sham+Telaglenastat), TAC surgery + vehicle(TAC+Vehicle), and TAC surgery + Telaglenastat(TAC+Telaglenastat). Cardiac function in the four groups was assessed by echocardiography. Hematoxylin-eosin(HE) staining and picrosirius red(PSR) staining were used to observe histopathological changes in heart tissues from the four groups. Results : CCK-8 assay results showed that, compared with the DMSO control group, there was no significant cytotoxicity in normal cardiomyocytes( H9C2 and AC16) treated with various concentrations of Telaglenastat(P>0. 05), except that a statistically significant difference in cell viability was observed in AC16 cells treated with 5 μ mol/L Telaglenastat(P<0. 001). In the PE-induced cardiomyocyte hypertrophy model, compared with the DMSO+PE group, the Telaglenastat+PE group showed significantly downregulated mRNA and protein expression levels of the cardiac hypertrophy markers Anp and Bnp(P<0. 01). In the TAC mouse model, compared with the TAC+Vehicle group, the TAC+Telaglenastat group exhibited significantly increased left ventricular ejection fraction(EF) and fractional shortening(FS)(P<0. 05), as well as significantly reduced heart weight-to-body weight ratio(HW/BW), lung weight-to-body weight ratio(LW/BW), and heart weight-to-tibia length ratio(HW/TL)(P<0. 01). HE and PSR staining results showed that, compared with the TAC+Vehicle group, the TAC+Telaglenastat group had smaller cardiomyocyte cross-sectional areas and less cardiac collagen fibrosis. Transcriptomic analysis revealed that, compared with the DMSO+PE group, the Telaglenastat+PE group exhibited downregulated expression of genes related to inflammatory response, fibrosis, and protein synthesis, along with upregulated expression of genes involved in the mitophagy pathway. Conclusion : Telaglenastat effectively inhibits pathological cardiac hypertrophy and improves cardiac function both in vitro and in vivo, and its protective effects may be associated with the activation of the mitophagy pathway in cardiomyocytes.
Causal association between circulating inflammatory cytokines and allergic diseases: a two-sample Mendelian randomization study
XIANG Youfu;XIONG Shiyin;QIU Xiufeng;LU Puyang;MIN Lingfeng;WU Xinhao;YOU Cong;The First Clinical Medical School of Gannan Medical University;Objective : To explore the causal relationship between inflammatory factors and atopic dermatitis(AD), allergic rhinitis(AR), and allergic asthma(AA) using a two sample Mendelian randomization(TSMR) analysis method. Methods : Data was collected on 41 inflammatory factors from a genome-wide association study(GWAS) as exposure and data on AD, AR, and AA from the finnGen study as outcomes. The TSMR analysis, we employed the inverse-variance weighted(IVW) method, the weighted median(WM) method, and the MR-Egger regression method to infer causality. Several sensitivity analysis methods were used to assess the stability of the results, and the MR Steiger test was used to determine the correctness of the causal direction. Results : The TSMR analysis revealed that macrophage colony-stimulating factor(M-CSF) was associated with an increased risk of AD(IVW:OR=1. 053, 95%CI=1. 001-1. 107, P=0. 044), Interleukin-5 was associated with an increased risk of AR(IVW:OR=1. 078, 95%CI=1. 000-1. 161, P=0. 049), Regulated upon activation, Normal T cell expressed and secreted(RANTES) was associated with a reduced risk of AR(IVW:OR=0. 922, 95%CI=0. 856-0. 994, P=0. 034), and granulocyte colony-stimulating factor(G-CSF) was associated with a reduced risk of AA(IVW: OR=0. 880, 95%CI=0. 782-0. 990, P=0. 033). Sensitivity analyses confirmed the robustness of these results, and the MR Steiger test did not detect reverse causality. Conclusion : M-CSF may be an upstream risk factor for atopic dermatitis, IL-5 may be an upstream risk factor for allergic rhinitis, RANTES may be an upstream protective factor for allergic rhinitis, and G-CSF may be upstream protective factors for allergic asthma.
Experimental Study on Anti-inflammatory effect of Osthol
Lian Qishen; Zhou Li; Ye Heyang; et alOsthol(Ost) was a coumarin isolated from the fruits of Cnidium monnieri(L ) Cusson, Ostsuppressed the swelling of mouse ears induced by zylene and the increase of peritoneal caPillarypermeability by ip 0.7% acetic acid in mice. Ost diminished the ederna of hind paws induced by sc1% carrageenan not only in normal rats, but also in adrenalectomised rats. However, the conentsof PGE in the inflammtory tissue of rat hind paws was not changed. Furthermore, Ost inhibitedthe proiferation of granulotna induced by impanting 0.1 ml agar in mice. These results suggestedthat Ost had anti-inflammatory effects, which was neither dependent on the pituitary-adrenalsystem nor on the PGE synthesis.
Effect of Pericarpium Citri Reticultate on gastric emptying and gastrointestinal propulsive motility in mice
HUANG Zhi hua,XIONG Xiao qing,et al (Department of Physiology, Gannan Medical College 341000)Objective and Methods: The purpose of this study was to exaluate the effect of Pericarpium Citri Reticultate on gastric emptying in rats and gastrointestinal propulsive motility in mice. Result: The result showed that 0.8ml of 20% Pericarpium Citri Reticultate decoction could obviously promote gastric emptying of rats, and inhibited gastrointestinal populsive motility of mice.
The Antibiosis Effects of Chinese Herbal Medicine Such as Schisandra Chinensis Baill etc on Enteric Pathogen and Conditioned Pathogen
MA Lian lan,LI Juan,LIU Zhi chun,WANG Xiao li,XIE Shui xiang (Gannan medical college,ganzhou,jiangxi,341000)Objective:To discuss the antibiosis effects of about 12 kinds of Chinese medicinal herbs,such as Schisandra Chinensis Baill etc,on Dysentery bacterium?S.Typhi?P.aruginoas?C.albicans.Methods:(1) The herbs were extracted by using 50% ethanal,by means of agar plate diffusions,observing the diameter size of circle of inhibition;(2) By agar plate continuously dilution oberserving the drugs of minimal inhibitive concentration (MIC,mg/ml).Results:(1)Five kinds of Chinese midicine herbs,such as Schisandra Chinese Baill?Rbus Chinensis Mill?Coptis Chinensis Franch?R.officinale Baill,have antibiosis effects against the above mentioned four sorts of experimental bacteria(The diameter size of circle of inhibition is between 13mmand 39mm);(2) Four kinds of Chinese medicine herbs such as Crataegus pinnatifida Bge.Fraxinus rhynchophylla Hance?Phyllanthus urinaria L.Acalypha australis L,have antibiosis effects on three sorts of experimental bacteria (The diameter size of circle of inhibition is between 12mm and 40mm);(3) Only Anemarrhena asphoddoidses Bunge has the antibiosis effects on C.albicans;(4)The difference of MIC about the above mentioned herbs is rather big on experimental bacteria,Schisandra chinensis Baill has the lowest MIC which is 6.25mg/ml on three sorts of bacteria;Coptis Chinensis Franch has the lowest MIC which is 5mg/ml on C.albicans.Conclusion:Schisandra Chinensis Baill Rhus Chinensis Mill?Coptis Chinensis Franch?Scutellaria Baicalensis Georgi?R.officinale Baill have the bacteriostatic on all experimental bacteria;Nine kinds of Chinese medicinal herbs such as Schisandra Chinensis Baill?Rhrs Chinensis Mill?Crataegus pinnatifida Bge.et al,have the antibiosis effects on Dysentery bacterium?S.typhi?P.aeruginosa;Six kinds of Chinese medicine herbs,such as Coptis chinensis Franch?Anemarrhena asphodeloidses Bunge?Rhus chinensis Mill et al,have antibiosis effects on C.albicans;Schisandra chinensis Baill has the best antibiosis effects on intestine-path pathogen and conditioned bathogen,which the biggest diameter size of circle of inhibitory is 40mm,the lowest MIC is 6.25mg/ml;Coptis chinensis Franch has the best antibiosis effects on C.albicans,which the biggest diameter size of circle of inhibition is 32mm,the lowest MIC is 5mg/ml.
External Bacteriostatic Experimentation About Twenty-nine Kinds of Chinese Medicinal Herbs such as Schisandra Chinensis Baill,etc
LIU Zhi-chun,WANG Xiao-li,LIN Peng,et al (Gannan medical college,ganzhou,jiangxi,341000)Objective:To discuss bacteriostatic efficacy about twenty- nine kinds of Chinese medicinal herbs,such as Schisandra chinesis Baill etc, on S.aureas、S.flexneri、P.aeruginosa。Methods:(1) The herbs were extracted by using 50% ethanal,by means of agar plate diffusions observing the diameter size of circle of inhibition;(2)By agar plate continuonsly dilution oberserving t he drugs of minimal inhibitive concentration (MIC,mg/ml)。Results:(1)Fifteen kinds of Chinese midicine herbs,such as Schisandra chinesis Baill、Rhus chinens is Mill、Scutellaria baicalensis Georgi、R.officinale Baill、Magnolia officinali s Rehd.et wils、Fraxinus rhynchophylla Hance etc,have bacteriostatic efficacy against the above mentioned three sords of experimental bacteria(The diameter s ize of circle of inhibition is between 12mmand 40mm);(2) Four kinds of Chines e me dicine herbs such as Acalypha australis L.、Salvia miltiorrhiza Bunge、Paeonia s uffruticosa Andr、Phellodendron Chinese schneid,have bacteriostatic efficacy on S.aureas and S.flexneri (The diameter size of circle of inhibition is between 1 2mm and 35mm),but not P.aeruginosa;(3)It has the bacteriostatic efficacy onl y against one sort of experimental bacteria such as Glycyrrhiza uralensis Fisch 、Gentiana maccrophylla pall、Polygonum multiflorum Thunb、paris poludhulla Smit h etc;(4)The difference of MIC about the above mentioned herbs is rather bi g on experimental bacteria,Schisandra chinensis Baill has the lowest MIC which is between 6.25mg/ml and 10.00mg/ml on three sorts of bacteria.Conclusion:Fifte en kinds of Chinese midicine herbs,such as Schisandra chinesis Baill、Rhus chin ensis Mill、Scutellaria baicalensis Georgi、R.officinale Baill、Magnolia officin alis Rehd.et wils、Fraxinus rhynchophylla Hance etc, have the bacteriostatic efficacy on all experimental bacteria;Acalypha australis L.、Salvia miltiorrhiz a Bunge、Paeonia suffruticosa Andr、Phellodendron Chinese schneid have the bacte riostatic efficacy on S.aureas and S.flexneri;Schisandra chinensis Baill which the biggest diameter size of circle of inhibitory is 40mm and the lowest MIC is 6.25mg/ml,has the best bacteriostatic efficacy on three kinds of experimental b acteria.
Advances in the treatment of acne
WU Wei;LI Yan;ZENG Zhao-lin;Gannan Medical University;Acne is a chronic disease of the sebaceous gland unit, presenting as inflammatory or non-inflammatory changes that mainly affect the face, and also the back and chest. At present, the pathogenesis of acne has not been thoroughly studied.Current data indicate that there are many pathogenesis factors of acne, including genetic factors,excessive sebum secretion, abnormal keratosis of sebaceous ducts in hair follicles, massive multiplication of propionibacterium acne, inflammation and immune reaction, etc. The commonly used treatment methods for acne are topical drug therapy, systematic drug therapy, photoelectric therapy,chemical stripping therapy,microneedle therapy, etc. This article reviewsthe research progress in thetreatment of acne.
Current situation and progress of rehabilitation treatment of autism
QU Jia-yang;HE Zheng-yi;LI Lin-cai;ZOU Zheng-wei;WANG Mao-yuan;YE Jun-son;Gannan Medical University;Autism spectrum disorder is a neurodevelopmental disorder characterized by repetitive behaviors,social dysfunction,and interest stenosis. The pathogenesis of autism has not been clear up to now,and there is no specific medicine. The increasing number of people with autism in China has brought great pressure to the family and society.Appropriate treatment will improve the core symptoms of autistic children,and it is very important to choose the appropriate method among many intervention means. However,at present,there is chaos and problems in the autism rehabilitation field,and the professionalism of various institutions in the rehabilitation field is uneven. This paper will discuss and summarize several intervention methods and their effects. At present,in addition to the conventional treatment methods utilized by rehabilitation institutions,speech therapy,sentiment training,behavior intervention and structured training,art therapy,peer support therapy and animal-assisted therapy are also gradually emerging,making up for the shortcomings of traditional training methods. As a new approach to treat autism based on cell technology,stem cell therapy for autism is one of the hot spots in current autism treatment research,which is expected to effectively improve the core symptoms of autism. Given the heterogeneity of autism,a single treatment is not desirable. The injection therapy of stem cells and the intervention of various rehabilitation treatment methods will be a new model of autism treatment in the future.
Progress in the mechanism of aflatoxin toxicity
ZHENG Yu-yuan;LIN Fen;QIU Wei;TAN Xiao-wei;Gannan Medical University;Aspergillus producing aflatoxins are widely distributed in nature and seriously pollute the food supply of human beings and animals,causing health hazards and even death. Therefore,aflatoxins has become the focus of attention in the current food safety field. This article briefly introduces the molecular structure,physicochemical properties,metabolism,and hazards and limitations of aflatoxins,and focuses on the more common signal pathways and their molecular biology mechanisms that cause liver cancer,providing the theoretical basis for protecting consumers' health.
Research progress on pathogenesis of Parkinson's disease
YOU Jing;GU Qiao-qiao;YU Zi-yun;ZHU Jiang-yang;TANG Kun-xin;WEN Er-sheng;HUANG Zhi-hua;XUE Jin-hua;CHEN Tao;Parkinson's disease(PD) is a neurological disease that occurs in middle-aged and elderly people and is the second most common neurodegenerative disease after Alzheimer's disease. The main pathological feature of Parkinson's disease is progressive degeneration and necrosis of dopaminergic neurons in the substantia nigra pars compacta, resulting in a decrease in dopaminergic(DA) neurotransmitters in the nigrostriatal pathway. The pathogenesis of PD is complex. It is currently believed to be the result of genetic, environmental, and ageing factors. The specific mechanism may involve mitochondrial dysfunction, oxidative stress, and neuroinflammation. This article is intended to review the research on the pathogenesis of PD in recent years.
An investigation on the take-out behavior of college students in Ganzhou and its influencing factors
YANG Hong;LIU Yu-jie;LUO Hui-juan;CHEN San-qian;WEI Yi-hui;WANG Yu-ting;JIA Xin-hua;WANG Qi;Gannan Medical University;Objective: To investigate the current situation of take-away behaviors of college students in Ganzhou, to understand the average number of take-outs per week, the safety awareness of take-away and the awareness of its nutritional value, and to provide suggestions for improvement by analyzing the possible factors affecting the average number of take-outs per week. Methods: A questionnaire survey was conducted among college students from 3 universities in Ganzhou by random sampling. The data were analyzed by descriptive analysis, univariate analysis and ordinal logistic regression. Results:(1)21.1% college students think that take-away food is unsafe or highly unsafe, and 68.1% think it is normal, but 10.9% think that take-away food is safe or highly safe. 26.9% college students disapproved or obviously disagreed with the nutritional value of take-away foods,and 66.4% thought it was normal, but 6.7% of them agreed or strongly agreed that the take-away foods has nutritional value.(2)Among the average number of take-aways per week in the last three months, 40.6% have not ordered take-aways, 36.2% have been 1-2 times a week, 10.9% have been 3-4 times a week,5.4% have been 5-6 times a week and 6.9% for 6 times a week and above.The results of ordinal logistic regression analysis showed that gender, profession, grade, monthly basic living expenses, cognitive level of nutritional value of take-outs, and the increase in economic burden of takeaways were the influencing factors of the number of take-outs. Conclusion: The understanding of the health quality and nutritional value of take-away foods is not comprehensive. College students should actively acquire knowledge about take-away foods safety and nutritional value, and raise the level of relative knowledge so as to reasonably choose take-away foods.
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Supervisor: Gannan Medical University
Sponsor: Gannan Medical University
Edited and Published by: Editorial Office of Journal of Gannan Medical University
Editor-in-Chief: ZHANG Xiaokang
Address: No. 1 Hexie Avenue, Rongjiang New District, Ganzhou, Jiangxi Province, China
Tel: 0797-8169729
Distribution: Publicly Distributed at Home and Abroad
E-mail: gnyxxb@vip.163.com
First Published: 1980
Publication Frequency: Monthly
ISSN: 2097-7174，CN: 36-1357/R
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